Cloves stop growth of multiple types of cancers

Cloves stop growth of multiple types of cancers

Cloves are a potent spice that have been used for centuries in traditional medicine to heal various ailments. They have antiviral, antibacterial, and antifungal properties, and have also been known as a pain reliever. New research shows that cloves stop growth of multiple types of cancers.

The researchers tested a variety of cancer cells in laboratory and treated them with clove extracts. The researchers tested ovarian cancer cells, liver cancer cells, colon cancer, breast cancer and pancreatic cancer cells.

The researchers found that the clove extract stopped the activity and growth of the colon cancer, breast cancer, ovarian, and liver cancer cells.

The cloves were also not dangerous to normal cells. They inhibited the growth of cancer cells, while leaving normal healthy cells in tact.

As written in the study abstract:

The results showed that the proliferation of Panc-28 cells was inhibited by OA in a concentration-dependent manner, with an IC50 (The half maximal inhibitory concentration) value of 46.35 µg ml−1, as determined by MTT (3-(4,5-Dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide) assay. The cell cycle was arrested in S phase and G2/M phase by OA.”

In other words, the effect of cloves on cancer cells was dose-dependent. This means that the more clove extract was applied, the stronger the anti-cancer effect. The dose-dependent factor is one of the most important when judging the cancer-fighting properties of a compound.

The study showing that cloves stop growth of multiple types of cancer was published in the Journal of Applied Toxicology.

Note: None of the information in our website is intended to diagnose, treat, cure or prevent any illness or disease. The content on our website is for educational purposes only.

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REFERENCES:
1. “Cloves Stop the Growth of Several Cancers In Preclinical Research.” GreenMedInfo.com. GreenMedInfo.com, n.d. Web. 07 Dec. 2015.
2. “Oleanolic Acid Arrests Cell Cycle and Induces Apoptosis via ROS-mediated Mitochondrial Depolarization and Lysosomal Membrane Permeabilization in Human Pancreatic Cancer Cells.” Wiley. Journal of Applied Toxicology, n.d. Web. 07 Dec. 2015.

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